Dysmetabolic Nephropathy

Primary and secondary dysmetabolic nephropathy is allocated depending on the causes.

In the first case, the disease ishereditary and is characterized by development in the early stages of urolithiasis and progressive course. There is also a development of chronic kidney failure.

Secondary dysmetabolic nephropathy may beis associated with excessive intake of any substances in the body or with diseases of the gastrointestinal tract. The most common secondary dismetabolic nephropathy.

Diseases in the form of oxalate-calciumNephropathy is more common in children. The cause of the occurrence is, as a rule, a violation of the exchange of oxalates and calcium. The causes of oxalate formation can be intestinal anastomoses, ulcerative colitis, Crohn's disease, and excessive intake of oxalates with food. Oksalatnaya dismetabolicheskaya nephropathy in children in most cases is hereditary. An important role, in addition to genetic factors, is played by an unfavorable ecological situation, unbalanced nutrition and stresses. The first signs can appear even in newborns, but are more often detected at the age of 6-7 years. For children with this disease are characterized by vegeto-vascular dystonia, obesity, allergies. The overall development of the child with this disease does not suffer. Progression of nephropathy can lead to the development of urolithiasis, and with the layering of bacterial infection - to inflammation of the kidneys.

Phosphate dismetabolic nephropathy isa sign of a violation of phosphorus-calcium metabolism. The main cause of its occurrence are chronic infections of the urinary system. Urinary nephropathy is a violation of uric acid metabolism. A characteristic feature in this disease is urine of brick color.

Treatment of dismetabolic nephropathy isin the application of specific methods of therapy, the observance of diet and proper drinking regimen, in the overall normalization of the way of life. A universal method of treating nephropathy is the use of a large volume of fluid, since this reduces the concentration of substances soluble in urine. The intake of fluids before bedtime increases the volume of nighttime urination. Take preferably plain or mineral water. The goal of the diet is to reduce the salt load on the kidneys. Specific therapy is directed to normalization of metabolic processes, elimination of salts and prevention of crystal formation processes.

When treating oxalate nephropathy patientsprescribe cabbage and potato diet. The diet excludes strong meat broths, chillies, chocolate, cocoa, sorrel and spinach, carrots, beets and cranberries. Recommended use of pears, prunes and dried apricots. Medicinal therapy consists in the intake of antioxidants and membranotropic drugs. Treatment is long-term. It is prescribed vitamin B6 (pyridoxine) monthly course, as well as vitamin A, which normalizes the interaction of lipids and proteins of the cell membrane. Vitamin E (tocopherol acetate) is a powerful antioxidant and also strengthens the lipid-protein bonds of cell membranes. Used also as membrane stabilizers, xidiphon and dimephosphon. In the treatment of crystalluria, the efficacy of cystone and magnesium oxide is shown.

From the diet should exclude the richpurine bases products such as kidneys, liver, nuts, beans, peas. It is necessary to use mainly products of vegetable and milk origin. A good result is the reception of a large number of liquids, including decoctions of such herbs as horsetail, clover, sporich. Mineral water is slightly alkaline slightly mineralized.

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